OI not merely suppressed lipopolysaccharide-induced proinflammatory cascades of inducible nitric oxide synthase, cyclooxygenase-2, and cytokines launch in mouse BV2 microglial cells but in addition activated the Nrf2 signaling pathway and its downstream targets in these cells. Conditioned medium derived from OI-handled BV2 cells guarded from rotenone- and MPP+-induced neurotoxicity in https://genghisd443rdo6.ltfblog.com/profile